Introduction. Biofilm Formation in Response to the Gallbladder Environment S. Typhi secretes a pool of effectors into host cells through the SPI‐1 T3SS‐promoting invasion.
The minimum infectious dose is less than salmonella enteritis.
Typhoid fever is caused by Salmonella typhi bacteria. After passing through the intestinal wall is the lymphatic and hematogenous spread with secondary settlement in the spleen, liver, bone marrow, bile ducts, skin, and Peyer's patches.
I have read and accept the Wiley Online Library Terms and Conditions of UseThe burden of typhoid fever in low‐ and middle‐income countries: A meta‐regression approachA novel linear plasmid mediates flagellar variation in Combined high‐resolution genotyping and geospatial analysis reveals modes of endemic urban typhoid fever transmissionSalmonella SPI1 effector SipA persists after entry and cooperates with a SPI2 effector to regulate phagosome maturation and intracellular replicationPrecise excision of the large pathogenicity island, SPI7, in Bacterial guanine nucleotide exchange factors SopE‐like and WxxxE effectorsPseudogenization of the secreted effector gene sseI confers rapid systemic dissemination of National enteric disease surveillance: Salmonella annual reportReceptor‐mediated sorting of typhoid toxin during its export from Loss of very‐long O‐antigen chains optimizes capsule‐mediated immune evasion by Salmonella disrupts host endocytic trafficking by SopD2‐mediated inhibition of Rab7The typhoid toxin promotes host survival and the establishment of a persistent asymptomatic infectionHost adaptation of a bacterial toxin from the human pathogen salmonella typhiEvaluation of the clinical and microbiological response to The microbiological and clinical characteristics of invasive Bile acids function synergistically to repress invasion gene expression in Feverlike temperature is a virulence regulatory cue controlling the motility and host cell entry of typhoidal Enteric fever in Cambodian children is dominated by multidrug‐resistant H58 Invasive non‐typhoidal Salmonella disease: An emerging and neglected tropical disease in AfricaRapid emergence of multidrug resistant, H58‐lineage Intragenic recombination in a flagellin gene: Characterization of the H1‐j gene of A salmonella protein antagonizes Rac‐1 and Cdc42 to mediate host‐cell recovery after bacterial invasionCloning and molecular characterization of genes whose products allow Salmonella chronic carriage: Epidemiology, diagnosis, and gallbladder persistenceSseK1 and SseK3 type III secretion system effectors inhibit NF‐κB signaling and necroptotic cell death in Salmonella Typhi encodes a functional cytolethal distending toxin that is delivered into host cells by a bacterial‐internalization pathwaySalmonella enterica serovar‐specific transcriptional reprogramming of infected cellsThe innate immune response to bacterial flagellin is mediated by Toll‐ like receptor 5Direct nucleation and bundling of actin by the SipC protein of invasive Mechanisms to evade the phagocyte respiratory burst arose by convergent evolution in typhoidal Identification of GtgE, a novel virulence factor encoded on the Gifsy‐2 bacteriophage of Temporal fluctuation of multidrug resistant salmonella typhi haplotypes in the Mekong river delta region of VietnamSalmonella AvrA coordinates suppression of host immune and apoptotic defenses via JNK pathway blockadeIdentification of two targets of the type III protein secretion system encoded by the inv and spa loci of Cutting edge: Inflammasome activation in primary human macrophages is dependent on flagellinSseF and SseG are translocated effectors of the type III secretion system of SseK1 and SseK2 are novel translocated proteins of Virulence plasmid‐borne spvB and spvC genes can replace the 90‐kilobase plasmid in conferring virulence to Comparison of genome degradation in Paratyphi A and Typhi, human‐restricted serovars of Burden of typhoid fever in low‐income and middle‐income countries: A systematic, literature‐based update with risk‐factor adjustmentSipB‐SipC complex is essential for translocon formationThe C terminus of SipC binds and bundles F‐actin to promote Chronic typhoid carriage and carcinoma of the gallbladderDiscovery of novel secreted virulence factors from Identification of a pathogenicity island required for Complete genome sequence of a multiple drug resistant Composition, acquisition, and distribution of the Vi exopolysaccharide‐encoding GogB is an anti‐inflammatory effector that limits tissue damage during Predicting the impact of vaccination on the transmission dynamics of typhoid in South Asia: A mathematical modeling studyBroad detection of bacterial type III secretion system and flagellin proteins by the human NAIP/NLRC4 inflammasomeInhibition of nuclear transport of NF‐ĸB p65 by the Complementary activities of SseJ and SifA regulate dynamics of the So similar, yet so different: Uncovering distinctive features in the genomes of Differential bacterial survival, replication, and apoptosis‐inducing ability of Proteolytic targeting of Rab29 by an effector protein distinguishes the intracellular compartments of human‐adapted and broad‐host Virulence gene profiling and pathogenicity characterization of non‐typhoidal An extensive repertoire of type III secretion effectors in Reduced invasion to human epithelial cell lines of How to become a top model: Impact of animal experimentation on human Pseudogenization of sopA and sopE2 is functionally linked and contributes to virulence of Role of the viaB locus in synthesis, transport and expression of The Vi‐capsule prevents Toll‐like receptor 4 recognition of The Vi capsular polysaccharide prevents complement receptor 3‐mediated clearance of The flagellar regulator TviA reduces pyroptosis by Phylogeographical analysis of the dominant multidrug‐resistant H58 clade of SopE, a secreted protein of Salmonella dublin, is translocated into the target eukaryotic cell via a sip‐dependent mechanism and promotes bacterial entryThe emergence and outbreak of multidrug‐resistant typhoid fever in ChinaVying for the control of inflammasomes: The cytosolic frontier of enteric bacterial pathogen–host interactions, Activation of apoptosis by Salmonella pathogenicity island-1 effectors through both intrinsic and extrinsic pathways in Salmonella-infected macrophages, Journal of Microbiology, Immunology and Infection, Salmonella enterica Serovar Typhi exposure elicits deliberate physiological alterations and triggers the involvement of ubiquitin mediated proteolysis pathway in Caenorhabditis elegans, International Journal of Biological Macromolecules,
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Salmonella is a Gram-negative, rod-shaped, motile bacilli which move with the use of its peritrichous flagella.The genus Salmonella can be divided into two species (S. enterica and S. bongori), based on their phenotypic profile.The genus Salmonella is a member of the family Enterobacteriaceae.
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Typhoid fever is an infectious disease of global distribution. Your message goes here Salmonella Typhi pathogenesis Salmonella Typhi pass through fecal contaminated food or drinking water in the gastrointestinal tract. Ledeboer NA, Frye JG, McClelland M, Jones BD.
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Pathoadaptive Alteration of
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